Biological Sciences, School of
School of Biological Sciences: Faculty Publications
Accessibility Remediation
If you are unable to use this item in its current form due to accessibility barriers, you may request remediation through our remediation request form.
Document Type
Article
Date of this Version
2013
Citation
THE JOURNAL OF BIOLOGICAL CHEMISTRY VOL. 288, NO. 28, pp. 20435–20442, July 12, 2013
Abstract
Background: Host innate immunity is against virus infection and replication.
Results: Toll-like receptor 3 activation leads to enhanced expression of a key Kaposi’s sarcoma-associated herpesvirus (KSHV) protein.
Conclusion: KSHV uses host Toll-like receptor pathway to augment its critical gene expression. Significance: A virus may usurp host innate immunity for its own benefits.
Comments
© 2013 by The American Society for Biochemistry and Molecular Biology, Inc.