Off-campus UNL users: To download campus access dissertations, please use the following link to log into our proxy server with your NU ID and password. When you are done browsing please remember to return to this page and log out.
Non-UNL users: Please talk to your librarian about requesting this dissertation through interlibrary loan.
EFFECTS OF HALOPERIDOL UPON RESPIRATION AND OXIDATIVE PHOSPHORYLATION BY RAT BRAIN MITOCHONDRIA AND, HALOPERIDOL ANTAGONISM TO THE LETHAL UNCOUPLING EFFECTS OF 2,4-DINITROPHENOL
Abstract
The mechanisms by which drugs evoke pharmacologic responses in living organisms are not well understood. Normal circumstances the oxidative metabolism of living cells leads to the synthesis of compounds containing high-energy phosphate bonds which are known to be essential for such gross functions as growth, muscle contraction, nerve conduction, and, to certain specific functions such asmaintaining gradients of particular cations and anions to the translocation of others. It has been demonstrated that some drugs inhibit the synthesis, or the utilization, of high-energy phosphate compounds.
Subject Area
Pharmacology
Recommended Citation
GATZ, EDWARD ERWIN, "EFFECTS OF HALOPERIDOL UPON RESPIRATION AND OXIDATIVE PHOSPHORYLATION BY RAT BRAIN MITOCHONDRIA AND, HALOPERIDOL ANTAGONISM TO THE LETHAL UNCOUPLING EFFECTS OF 2,4-DINITROPHENOL" (1968). ETD collection for University of Nebraska-Lincoln. AAI7012262.
https://digitalcommons.unl.edu/dissertations/AAI7012262